Although thyroid diseases affect millions of Americans, few understand its function and just how important it is to our overall health, wellness and vitality.
When most people think of the thyroid, the first thing that may come to mind is an image of a simple, obscure gland located somewhere in the neck. The thyroid is often seen as serving unknown functions as it is rarely discussed in popular press. Likewise, it’s not at all surprising that few understand its exact function and just how important it is to our overall health, wellness and vitality.
In this article we will briefly cover what the thyroid is and its functions, and discuss the 5 most common thyroid disorders today.
The thyroid is an endocrine gland that produces and directs various hormones in the body. The health of the thyroid is paramount to overall well being, playing a vital role in cell growth, metabolism, and energy levels. The thyroid produces two major primary hormones called triiodothyronine (T3) and thyroxine (T4). T3 is the more bio active version of the hormone, while T4 is considered the less active, storage form. Surprisingly, the thyroid outputs roughly 20 times more T4 than T3.
Lesser known, the thyroid’s secondary role is the production of the calcium-regulating hormone calcitonin, which regulates and balances blood calcium levels and calcium deposition in bones.
Thyroid-stimulating hormone (TSH) released from the pituitary helps regulate the hormonal output and balance of the thyroid, regulating how much of the primary T3 and T4 hormones are manufactured and released. Before all of that happens, the TSH release is first stimulated by the area of the brain that controls neuroendocrine and central nervous system function. The hypothalamus then sends out its own stimulatory hormone called thyrotropin-releasing hormone (TRH).
The 5 Most Common Thyroid Disorders
Thyroid disorders affect millions of Americans yearly, and the number of people afflicted continues to grow with each passing year. Growing research data suggests that aside from the genetic origin of the various clinically significant thyroid disorders, much of the causation is likely due to lifestyle factors such as constant exposure to a toxic environment, the consumption of chemically-laden food and water, as well as a deficiency of certain nutrients.
Though thyroid disorders affect men and women alike – up to 20 million Americans – the predominance of disorders are found in women, upwards of 80%. 1 in 8 women will experience some type of thyroid disorder in their lives. 
Hyperthyroidism is defined as an overactive thyroid gland which produces an overabundance of T3/T4 hormones. Symptoms of hyperthyroidism include goiter, heart palpitations, anxiety, excess sweating, diarrhea, weight loss, and muscle weakness. Causes are as diverse as its symptoms, but nevertheless are important to understand. Autoimmunity of the thyroid often leads to Grave’s Disease, a disorder that results in an overactive thyroid. Also, nodule formation and/or goiter formation in the thyroid, leading to inhibition of necessary hormone feedback loops, contributes to excess production of thyroid hormones. Excess dietary iodine intake can also increase risk for hyperthyroidism.
Conventional approaches to hyperthyroidism include beta blockers, radioactive iodine, and surgery. Natural approaches are numerous and often boil down to one thing: diet. Eliminating goitrogenic foods may be helpful, as would removing fluoride, bromine, and chlorine from water via a high-quality filtration system. Reducing dietary gluten and dairy casein may also help protect the thyroid gland in sensitive individuals. Nascent iodine, lithium orotate, probiotics, vitamin D3, omega -3 fats, L -dopa (mucuna pruriens), and L-tyrosine are possible helpful supplements that can be taken for supporting thyroid health. Make sure to get plenty of sleep to recharge the thyroid, and avoid synthetic chemicals whenever possible. Deep breathing meditation and general relaxation may also be helpful for reducing stress associated with the thyroid.  
On the opposite end of the spectrum, an underactive thyroid which produces inadequate amounts of T3/T4 thyroid hormones is defined as hypothyroidism. Symptoms include tiredness, excessive weight gain, cold intolerance, baldness, depression, dry skin/hair/nails, and irritability. Common causes include a congenital abnormality (thyroid deficiency from birth), autoimmune inflammation of the thyroid such as Hashimoto’s thyroiditis, nutritional iodine deficiency, TSH hormone deficiency via pituitary gland abnormality, heavy metal toxicity, and dysbiosis (imbalance of good vs. bad bacteria).
Common treatment is the injection of synthetic thyroid hormone called Levothyroxine to boost hormone levels. With the exception of increasing exercise, the natural action steps to reduce risk for hypothyroidism are exactly the same for hyperthyroidism.  Exercise may help boost thyroid hormones, providing support for a sluggish, underactive gland.
3. Hashimoto’s Thyroiditis
Hashimoto’s thyroiditis is an autoimmune disorder whereby the thyroid gland is attacked by the immune system in response to antibodies produced by exposure to an allergen. This reacts with the cells and tissues of the thyroid, causing inflammation and destruction of the gland, ultimately leading to hyperthyroidism followed by hypothyroidism. Fatigue, cold intolerance, constipation, goiter, weight gain, paleness/puffiness in face, sleepiness, joint/muscular pain, dry/brittle hair, and depression are common symptoms. 
Medical experts postulate that viruses, bacteria, and other foreign substances initiate the process of Hashimoto’s thyroiditis autoimmunity. Iodine deficiency in conjunction with fluoride/chlorine/bromine exposure may also be a contributing factor. A gluten allergy may be another hidden culprit behind Hashimoto’s disease. Vitamin D deficiency and dysbiosis are also common concerning factors. Conventional and natural approaches are similar to that of hypothyroidism.
4. Grave’s Disease
Similar to Hashimoto’s disease, Grave’s disease is an autoimmune disorder whereby the thyroid gland is attacked by the immune system from antibodies produced in response to an allergen. This confuses the cells of the thyroid, causing inflammation and the overproduction of T3/T4 thyroid hormones, eventually leading to an overactive thyroid (hyperthyroid). 
Symptoms include anxiety, heart palpitations, goiter, hand tremors, weight loss, insomnia, irritability, muscle weakness, diarrhea, heat intolerance, and eye problems. The causes are very similar to that of Hashimoto’s disease. Approaches are generally the same as with general hyperthyroidism and include beta blockers, anti -thyroid medications such as methimazole and propylthiouracil, radioactive iodine , surgery, and avoiding goitrogenic foods.
Thyroiditis is defined as the swelling or inflammation of the thyroid gland. There are a few main types:
- Hashimoto’s thyroiditis
- Postpartum thyroiditis potentially caused by autoimmune response, often in two phases:
- The first phase occurs months 1 through 4 postpartum, resulting in hyperthyroidism.
- The second phase generally lasts from months 4 through 8 postpartum and often results in a hypothyroid condition due to the exhaustion of thyroid hormones in the earlier phase. Recovery usually happens naturally 12- 18 months postpartum.
- Silent/painless thyroiditis, similar to postpartum but not related to birth.
- Subacute thyroiditis, similar to the others but causes pain in jaw/neck/ear, possibly from autoimmunity or infection.
Iodine deficiency in conjunction with fluoride/chlorine/bromine displacement may be an important contributing factor in thyroiditis. Gluten allergy, vitamin D deficiency, and dysbiosis may also be factors associated with the condition. Depending on the type of thyroiditis, medications usually vary depending on whether it presents initially with hyper - or hypothyroidism. 
– Dr. Edward F. Group III, DC, ND, DACBN, DCBCN, DABFM
- American Thyroid Association. Thyroid Disease. Fact Sheet.
- Delitala G, Masala A, et al. Plasma prolactin response to L dopa TRH and metaclopramide in thyrotoxicosis. Biomedicine. 1976 Jul;25(5):173 6.
- NIH/NEMDS. Hyperthyroidism. National Institute of Health. Fact Sheet.
- JNIG/NEMDS. Hypothyroidism. Fact Sheet.
- NIH/NEMDS. Hashimoto’s Disease. Fact Sheet.
- NIH/NEMDS. Grave’s Disease. Fact Sheet.
- Office of Women’s Health/U.S. HHS. Thyroid disease. Fact Sheet.
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